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2006). Furthermore, ku and sbcC are epistatic one to another, (Weller and Doherty 2001; Mascarenhas et al. 2006). In contrast, the addAB and recN mutations render cells sensitive and very sensitive to ionizing radiation and MMC, respectively, and the ku and sbcC mutations, markedly increase the sensitivity of recN or addAB cells to ionizing radiation and to MMC addition (Mascarenhas et al. 2006; Table 3).
Mol Microbiol 57:1074-1085 Robu ME, Inman RB, Cox MM (2001) RecA protein promotes the regression of stalled replication forks in vitro. Proc Natl Acad Sci USA 98:8211-8218 Robu ME, Inman RB, Cox MM (2004) Situational repair of replication forks: Roles of RecG and RecA proteins. J Biol Chem 12:10973-10981 Rocha EP, Cornet E, Michel B (2005) Comparative and evolutionary analysis of the bacterial homologous recombination systems. PLoS Genet 1:e15 Rupp (1996) DNA repair mechanisms. D. R (ed). American Society of Microbiology Press, Washington DC, pp 2277-2294 Rupp WD, Howard-Flanders P (1968) Discontinuities in the DNA synthesized in an excision-defective strain of Escherichia coli following ultraviolet irradiation.
2006; Table 2). g. g. g. g. ligD, ku, topB, mutS2, sbcC2 and sbcD2 genes). g. Stpahylococcus aureus) the smc and mutS2 genes seem to be essential (Forsyth et al. g. in S. pneumoniae) the ruvA, ruvB and recU genes, classified within the ε epistatic group, seem to be essential (Thanassi et al. 2002). Unlike in B. subtilis and in S. aureus cells (Petit and Ehrlich 2002), the conserved pcrA gene is not essential in S. pneumoniae (Forsyth et al. 2002; Thanassi et al. 2002). Naturally transformable bacteria (B.